00:00What is the role of cytomegalovirus in systemic lipos erythematos?
00:16In the past 10 years, we have discussed the role of cytomegalovirus in systemic lipos erythematos.
00:24In SLE, in layman language, it is an autoimmune disease that kills our immune system.
00:33If we had immune system, we would have infected our immune system.
00:39But this immune system affects our immune system.
00:43This is the most dangerous disease in SLE.
00:46As you can see, it is mostly female.
00:51The female hormone plays a role in the female hormone.
00:55At the same time, it is a role in the genetic system and the environment system.
00:59It is very important.
01:01In the past 10 years, we have seen a recent report in the publication of the publication of the American
01:07Society for lipos erythematos.
01:10This is an important finding.
01:13In the past 10 years, we have seen individuals with CMD infection.
01:17If we have an influenza infection, we have seen the maximum potential for 2% of people with cells.
01:24However, with the prevalence of infection, our population is in normal population around 70% to 80%.
01:32However, you can see that as we face a number of cells, there are 95% of cells.
01:36In our analysis, the chances of the development of the SLE are higher than the SLE.
01:43In our analysis, we have seen the EBV infection, which is a major role in SLE.
01:55In our analysis, we have seen the CMV infection and EBV infection.
01:59In this case, we have seen the normal and healthy individuals around 80% of the individuals.
02:06If we know that we have no infection, then we will measure the immunoglobulin and the virus and the DNA.
02:15We will know that we have no infection.
02:17If we know that we have no infection, we will produce the immune globulin.
02:22If we know that we have no infection, then we will have no infection.
02:27In this case, we will prime the immune system to the immune system.
02:31Then we will take the immune system to the response.
02:35Sir, we have seen the CMV infection with the SLE association.
02:41Then let us find our screening response at a more person.
02:47Coming up on the SLE, what if you do not catch the immune system from a completely jakam치 evidence?
03:06We have a report in the population.
03:13The actual mechanism is good.
03:18We have a pathway or mechanism to know the pathway.
03:24We have to study different experiments.
03:28We have to check different antibody levels.
03:31check the viral load and I will try to study the association.
03:37Generally, most of the people who are healthy individuals, who are patients,
03:42the virus is nearly 80% of the population.
03:48So, we say that the virus is nearly 80% of the population,
03:53the virus is already infected.
03:55So, according to the media, there are no symptoms.
04:04The virus is nearly 80% of the virus.
04:07The virus is the same as the virus.
04:11The virus is the same as the virus.
04:13The virus is the same as the virus.
04:15The virus is the same as the virus.
04:24The virus is the same as the virus.
04:25It is also clear that the virus is not infected.
04:25Even the virus is the same as the virus.
04:27The virus is the same as the virus.
04:27We even get infected with the virus.
04:28We also get infected with the virus.
04:29We also get infected with the virus.
04:30It is a virus.
04:31We are sadly vaccinated.
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